There
is a lot of information on this page. However, we
are adding to this Research information almost every week. Come back on Monday or
Friday and see what is new. Like the brand new GliSODin research
monograph.
Just click
HERE.
Patents, Articles and Clinical Studies on SOD, SOD/Gliadin
(GliSODin®).
1. THE FIRST ORALLY EFFECTIVE
VEGETARIAN FORM OF SUPEROXYDE DISMUTASE (SOD) October 9 2004
: THE SUPEROXIDE DISMUTASE/GLIADIN ("GliSODin") PRODUCT,
Bernard DUGAS, CSO, PhD
A Health Promoting and Anti-Aging Nutraceutical ProductImmunity,
Oxidative stress & Pharmacology Immunity. Article covers:
Oxidative Stress,
Pharmacology, References
and A Health Promoting and
Anti-Aging Nutraceutical Product (click
Here for full text of article).
2.
From Enzyme
University.com,
SOD (Superoxide dismutase)/gliadin,
Information provided by National Enzyme Company,
Apr 27, 2004, 17:24
SOD (Superoxide dismutase)/gliadin catalyzes the destruction of
superoxide free radicals. SOD/gliadin is a uniquely vegetarian
source of highly absorbable SOD that works synergistically with
endogenous SOD to prevent the oxidative effects of superoxide.
Evidence suggests that antioxidant enzymes, such as SOD/gliadin,
can be useful supplements for anti-oxidant support,
anti-inflammatory support, immune support, and cardiovascular
support.
3. United States Patent
6,045,809, Postaire , et al. April 4, 2000
Pharmaceutical compositions containing a superoxide dismutase
Abstract
Novel pharmaceutical compositions that are particularly suitable
for oral administration of superoxide dismutases (SODs) and that
have good bioavailability and therapeutic efficacy. These
compositions are made up of a combination of a superoxide
dismutase and at least one lipids or proteins. The lipids are
exemplified by ceramides, and the proteins are exemplified by
prolamines or polymer films based on such prolamines. A
pharmaceutically acceptable vehicle can also be included in the
composition. (click
Here for full text of patent)
4. United States Patent 6,190,658,
McCord , et al. February 20, 2001 Genetically
modified manganese superoxide dismutase for treating oxidative damage
Abstract
This invention discloses a genetically modified manganese superoxide dismutase
nucleic acid molecule and protein. Also disclosed are recombinant molecules,
recombinant cells, therapeutic compositions and methods of using the modified
manganese superoxide dismutase to treat oxidative damage. (click
Here for full text of patent)
5.
United States Patent 6,030,611,
Gorecki , et al. February 29, 2000
Therapeutic SOD compositions and uses thereof
Abstract
Improved methods for production of recombinant
human superoxide dismutase and analogs thereof have been developed. Such
superoxide dismutase or analogs may be used to catalyze the reduction
of superoxide radicals thereby preventing or treating injury resulting
from the presence of such superoxide radicals. (click
Here for full text of patent)
6.
United States Patent 6,503,506,
Germano January 7, 2003
Nutrient therapy for immuno-compromised patients
Abstract
A nutritional supplement is taught for treating
chronic debilitating diseases such as HIV/AIDS to overcome conditions
of oxidative stress, decreased lean muscle mass, decreased energy production
(mitochondrial failure) and support immune function. It comprises orally
administrable superoxide dismutase (SOD), preferably SOD/GLIADIN, in combination
with other antioxidant/immune support components (Beta Glucans, Nucleotides,
Fruit Polyphenols); High Immunoglobin Whey; (undenatured whey), Ornithine
alpha ketoglutarate (OKG), Branched Chain Amino Acids and Glutamine to
reduce loss of lean muscle mass; and Coenzyme Q 10, D-Ribose and L-Carnitine
to provide energy support (decrease mitochondrial failure).
7. Influence of an Orally Effective SOD on Hyperbaric
Oxygen-related Cell Damage
CLAUS M. MUTHa,b,*, YVONNE GLENZa, MICHAEL KLAUSa, PETER
RADERMACHERa, GU¨ NTER SPEITb and XAVIER LEVERV (click
Here for full text of
article in PDF format)
8. Influence of an Orally Effective Superoxide Dismutase (GliSODin)
on Strenuous Exercise-Induced Changes of Blood Antioxidant
Enzymes and Plasma Lactate. Y Hong, S Hong, Y.H. Chang and
S. H. Cho. \Exhausting exercise is responsible for a significant
increase in blood TAS, SOD, GR and Lac....GliSODin induces a
significant change in oxidative statue and a significant
decrease in exercise induced lactate release. (click
Here for full text of
article in PDF format)
9. GliSODin Improves Quality of Life for Women. In a
study of 100 women, 87% niticed an overall improvement in the
quality of their skin and 73% an improvement in the quality of
their life. (click
Here for full text of
article in PDF format)
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Articles, References, Clinical Studies and Scientific Papers on
the
Therapeutic Properties of Pollen Extracts
(Click on the Underlined text for complete article)
7. Additional SOD Publications (click
here)
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Clinical
Studies of the Therapeutic Value of Green Tea Extracts
(Click on the Underlined text for complete article)
1. Green Tea and Blood Pressure /
Hypertension Research
A study published in the July 2004 issue of the Archives of
Internal Medicine found that among persons consuming tea
regularly for at least one year, the risk of developing high
blood pressure was 46% lower among those who drank ˝ cup to 2 ˝
cups per day, and 65% less among those consuming more than 2 ˝
cups per day.
Yang YC, Lu FH, Wu JS, Wu CH, Chang CJ. The protective effect of
habitual tea consumption on hypertension. Arch Intern Med. 2004
Jul 26;164(14):1534-40.
In another study, this one of rats bred not only have high
blood pressure but also to be prone to strokes, those rats given
green tea had significantly lower systolic and diastolic blood
pressure compared to controls, who received plain water. The
animals in this study, which was published in the January 2004
issue of the Journal of Nutrition, consumed the human equivalent
of 1 liter (1.1 quarts or a little more than 4 cups) of green
tea per day. Negishi H, Xu JW,
Ikeda K, Njelekela M, Nara Y, Yamori Y. Black and green tea
polyphenols attenuate blood pressure increases in stroke-prone
spontaneously hypertensive rats. J Nutr. 2004 Jan;134(1):38-42
Green Tea Research - Protects the Liver from Alcohol and
Other Harmful Chemicals
Alcohol metabolism results in the production of damaging free
radicals that can overwhelm the liver's supply of antioxidants,
resulting in liver injury. In a study published in the January
2004 issue of Alcohol in which rats were chronically intoxicated
with alcohol for 4 weeks, green tea prevented damage to their
livers. Ostrowska J, Luczaj W,
Kasacka I, Rozanski A, Skrzydlewska E. Green tea protects
against ethanol-induced lipid peroxidation in rat organs.
Alcohol. 2004 Jan;32(1):25-32.
2. Green Tea and Allergy Reduction Research
In a study printed in the Oct. 2002 issue of the Journal
of Agricultural and Food Chemistry, researchers in Japan
identified a compound in green tea (ECGC) that may block a key
cell receptor involved in producing an allergic response in
humans. American Chemical Society:
http://center.acs.org/
3. Green Tea and its Effects in Arthritis Research
Antioxidants in green tea may reduce the severity of
rheumatoid arthritis, according to a study from Case Western
Reserve University's School of Medicine published in the April
13 issue of the Proceedings of the National Academy of Sciences.
http://www.cwru.edu
Independent, published articles have been completed showing
the positive benefits of Green Tea in the area arthritis. Below
are a handful of articles conducted on the effects of the
components found within Green Tea:
Green tea polyphenol
epigallocatechin-3-gallate inhibits the IL-1 beta-induced
activity and expression of cyclooxygenase-2 and nitric oxide
synthase-2 in human chondrocytes.Ahmed S, Rahman A, Hasnain A,
Lalonde M, Goldberg VM, Haqqi TM.Department of Orthopedics, Case
Western Reserve University, Cleveland, OH 44106-4946, USA.
[Study #6]
Epigallocatechin-3-gallate
inhibits interleukin-1beta-induced expression of nitric oxide
synthase and production of nitric oxide in human chondrocytes:
suppression of nuclear factor kappaB activation by degradation
of the inhibitor of nuclear factor kappaB.
Singh R, Ahmed S, Islam N, Goldberg VM, Haqqi TM. Case Western
Reserve University, Cleveland, Ohio 44106-4946, USA.
[Study #7]
Catechins from green tea
(Camellia sinensis) inhibit bovine and human cartilage
proteoglycan and type II collagen degradation in vitro.
Adcocks C, Collin P, Buttle DJ.
Division of Genomic Medicine, University of Sheffield Medical
School, Sheffield S10 2RX, UK.
[Study #8]
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